TFE3 fusions escape from controlling of mTOR signaling pathway and accumulate in the nucleus promoting genes expression in Xp11.2 translocation renal cell carcinomas.. TFE3 fusions escape from controlling of mTOR signaling pathway and accumulate in the nucleus promoting genes expression in Xp11.2 translocation renal cell carcinomas.
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https://www.ncbi.nlm.nih.gov/bioproject/PRJNA1110249
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资源简介：
ChIP analysis demonstrated that TFE3 fusions could bind to the promoters of the target genes as a wild-type TFE3 protein. Overall design: Chip-seq for TFE3 in UOK120 cells and UOK109 cells
创建时间：
2024-05-10
