Transcription profiling of human T-ALL patients with molecular cytogenetic abnormalities (i.e. TAL1 (n=24), LMO2 (n=9), HOXA (n=5), HOX11/TLX1 (n=7), and HOX11L2/TLX3 (n=22)) reveals the recurrent SET-NUP214 fusion as a new HOXA activation mechanism in pediatric T-ALL
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资源简介：
Transcription profiling of human T-ALL patients with molecular cytogenetic abnormalities (i.e. TAL1 (n=24), LMO2 (n=9), HOXA (n=5), HOX11/TLX1 (n=7), and HOX11L2/TLX3 (n=22)) reveals the recurrent SET-NUP214 fusion as a new HOXA activation mechanism in pediatric T-ALL
创建时间：
2008-04-07
