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Transcriptomic Comparison of Neuronal Development Stages using Induced Pluripotent Stem Cells from Bipolar Disorder Patients

ChEBI2015-10-26 更新2026-07-12 收录

精神疾病分子机制

干细胞神经发育

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-74358 数据链接链接失效反馈

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资源简介：

Transcriptomic Comparison of Neuronal Development Stages using Induced Pluripotent Stem Cells from Bipolar Disorder Patients

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创建时间：

2015-10-26

抑郁症生物标志物

精神疾病分子机制

BackgroundMajor depressive disorder (MDD) is a prevalent mental health condition in which mitochondrial dysfunction and cellular senescence contribute to its pathogenesis. This study aims to identify

NIAID Data Ecosystem70

Genome-Scale Transcriptional Regulatory Network Models of Psychiatric and Neurodegenerative Disorders. Homo sapiens

精神疾病分子机制

调控网络分析

Transcriptional regulatory changes in the developing and adult brain are prominent features of brain diseases, but the involvement of specific transcription factors (TFs) remains poorly understood. We

NIAID Data Ecosystem40

Additional file 8 of Transcriptomics and miRNomics data integration in lymphoblastoid cells highlights the key role of immune-related functions in lithium treatment response in Bipolar disorder

精神疾病分子机制

sRNA-mRNA互作网络

Additional file 8 Supplementary Table 8. List of 2027 significant mature miRNA-mRNA interactions with p-value ≤0.05 and Pearson correlation index ρ ≤ − 0.5.

Figshare2022-10-28 更新30

TCF7L2 lncRNA: A Link between Bipolar Disorder and Body Mass Index through Glucocorticoid Signaling. TCF7L2 lncRNA: A Link between Bipolar Disorder and Body Mass Index through Glucocorticoid Signaling

精神疾病分子机制

lncRNA功能研究

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series

NIAID Data Ecosystem60

Mouse, Hippocampus, LC-MSMS - Omega-3 fatty acid deficiency disrupts endocytosis, neuritogenesis, and mitochondrial protein pathways in the mouse hippocampus

神经细胞蛋白质组学

精神疾病分子机制

Omega-3 fatty acid (n-3 FA) deficiency is an environmental risk factor for schizophrenia, yet characterisation of the consequences of deficiency at the protein level in the brain is limited. We aimed

NIAID Data Ecosystem30

Transcriptomic Comparison of Neuronal Development Stages using Induced Pluripotent Stem Cells from Bipolar Disorder Patients

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