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A case study of a long-term glioblastoma survivor with a hypermutated genotype and recurrence.
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创建时间：
2019-05-08
相关数据集
Additional file 2 of O-GlcNAcylation protein disruption by Thiamet G promotes changes on the GBM U87-MG cells secretome molecular signature
Additional file 2. List of proteins identified with UniPotID.
Figshare2021-04-26 更新40
Additional file 1 of SWIM tool application to expression data of glioblastoma stem-like cell lines, corresponding primary tumors and conventional glioma cell lines
Table. GBM differentially expressed genes for Schulte et al. dataset [25]. The table reports the list of the statistically significant (adjusted p-value <0.05) DEGs in Official Gene Symbols identif
Figshare2018-11-30 更新20
Supplementary Data 3 from Correlation of Somatic Mutation and Expression Identifies Genes Important in Human Glioblastoma Progression and Survival
Supplementary Data 3 from Correlation of Somatic Mutation and Expression Identifies Genes Important in Human Glioblastoma Progression and Survival
NIAID Data Ecosystem10
Spatio-temporal evolution of the primary glioblastoma genome
Tumor recurrence following treatment is the major cause of mortality for glioblastoma multiforme (GBM) patients. Thus, insights on the evolutionary process at recurrence are critical for improved pati
NIAID Data Ecosystem30
Additional file 4 of Whole-genome sequencing of glioblastoma reveals enrichment of non-coding constraint mutations in known and novel genes
Additional file 4: Table S3. List of Key GBM genes (n = 78), pooled from SweGBM-1 FMGs and TCGA-GBM SMGs.
Figshare2020-06-09 更新20
