Additional file 1 of Lack of food intake during shift work alters the heart transcriptome and leads to cardiac tissue fibrosis and inflammation in rats

Additional file 1: Table S1. Fpkm data, analysis of rhythmicity and gene expression levels (related to Figs. 2, 3, and S3). Table S2. dryR analaysis of differential rhythmicity in gene expression (related to Fig. 2). Table S3. KEGG pathway analysis, rhythmically expressed genes (related to Fig. 2). Table S4. List of genes being rhythmically expressed in each enriched KEGG pathway (related to Fig. 2 and S2). Table S5. KEGG pathway analysis, differentially expressed genes (related to Fig. 3). Table S6. List of DNase I hypersensitive sites in rat heart and genomic location (related to Fig. 4). Table S7. Motif analysis for genes being up-regulated or down-regulated in WRF rats (related to Fig. 4). Table S8. Motif analysis for genes being rhythmically expressed in C, W, and WRF rat hearts (related to Fig. S5). Table S9. Results statistical analysis for Fig. 5A (related to Fig. 5). Table S10. KEGG pathway analysis of the genes assigned to the modules M0 to M37 (related to Fig. 5). Table S12. Results statistical analysis for Fig. 5B (related to Fig. 5). Table S13. Results statistical analysis for Fig. 5B (related to Fig. 5). Table S14. Quantification of picrosirius red staining (related to Fig. 6). Table S15. Quantification of CD45, CD68, and Collagen 1 staining (related to Fig. 6). Table S16. Summary of RNA-Seq library size and percentage alignment (related to Methods).