Targeting TCA cycle through Cuproptosis confers Synthetic Lethality to ARID1A Deficiency Hepatocellular Carcinoma [ATAC-Seq]
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https://www.ncbi.nlm.nih.gov/sra/SRP429150
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资源简介：
To further explore the mechanisms of glucose metabolic reprogramming in ARID1A deficiency HCC, we conducted multi-omics profilings in ARID1A deficient and wild type Hep3B cells. Overall design: ATAC-seq was performed to determine chromatin accessibility in ARID1A deficient and wild type Hep3B cells.
创建时间：
2023-09-08
