Additional file 1 of Transcriptomic analysis of brain tissues identifies a role for CCAAT enhancer binding protein β in HIV-associated neurocognitive disorder

Additional file 1: Table S1. Related to Figure 2. Full differential gene expression (DEG) between Minor Neurocognitive Disorder and HIV+ cognitively normal subjects (CNHIV+). Table S2. Related to Figure 2. Enriched cellular components between Minor Neurocognitive Disorder and HIV+ cognitively normal subjects (CNHIV+). Table S3. Related to Figure 2. Enriched biological processes between Minor Neurocognitive Disorder and HIV+ cognitively normal subjects (CNHIV+). Table S4. Disease-gene associations for differentially regulated genes in Minor Neurocognitive Disorders (MND) compared to HIV+ cognitively normal subjects (CNHIV+). Table S5. List of differentially regulated transcription factors and along with targets also differentially expressed in Minor Neurocognitive Disorder compared to HIV+ cognitively normal subjects (CNHIV+). Table S6. Related to Figure 2. All enriched pathways for C/EBPβ targets in Minor Neurocognitive Disorder compared to HIV+ cognitively normal subjects (CNHIV+). Table S7. Related to Figure 3. List of astrocyte specific marker genes that are also C/EBPβ targets in Minor Neurocognitive Disorder compared to HIV+ cognitively normal subjects (CNHIV+). Table S8. Related to Figure 3. All enriched pathways for C/EBPβ regulated astrocyte marker genes targets in Minor Neurocognitive Disorder compared to HIV+ cognitively normal subjects (CNHIV+).