IL-1beta activates IL-17 via calcium

NIAID Data Ecosystem2026-05-26 收录

肺炎链球菌感染免疫

NLRP3炎症通路

数据链接：

https://www.ncbi.nlm.nih.gov/sra/SRP156501 数据链接链接失效反馈

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IL-17A production via NLRP3 inflammasome-dependent IL-1Ã secretion aginst Pneumococcal inflammation in mice Overall design: mice infected with 2x10^8 CFU of S. pneumoniae

创建时间：

2019-02-23

The Lectin Pathway of Complement Activation Is a Critical Component of the Innate Immune Response to Pneumococcal Infection

补体系统免疫

肺炎链球菌感染免疫

The complement system plays a key role in host defense against pneumococcal infection. Three different pathways, the classical, alternative and lectin pathways, mediate complement activation. While th

Figshare2016-01-19 更新40

A2E Induces IL-1ß Production in Retinal Pigment Epithelial Cells via the NLRP3 Inflammasome

年龄相关性黄斑变性发病机制

NLRP3炎症通路

Aims With ageing extracellular material is deposited in Bruch’s membrane, as drusen. Lipofuscin is deposited in retinal pigment epithelial cells. Both of these changes are associated with age related

NIAID Data Ecosystem20

DataSheet1_Saikosaponin d Alleviates Liver Fibrosis by Negatively Regulating the ROS/NLRP3 Inflammasome Through Activating the ERβ Pathway.docx

纤维化机制

NLRP3炎症通路

Background and aims: Saikosaponin d (SSd) has a steroidal structure and significant anti-inflammatory effects. The purpose of this study was to explore the mechanism underlying SSd’s inhibitory effect

NIAID Data Ecosystem10

IL-1β Production through the NLRP3 Inflammasome by Hepatic Macrophages Links Hepatitis C Virus Infection with Liver Inflammation and Disease

肝炎病毒机制

NLRP3炎症通路

Chronic hepatitis C virus (HCV) infection is a leading cause of liver disease. Liver inflammation underlies infection-induced fibrosis, cirrhosis and liver cancer but the processes that promote hepati

NIAID Data Ecosystem30

ALKBH8 suppresses oxidative stress and ameliorates inflammation via blocking NF?B/NLRP3 axis in Sepsis

脓毒症分子机制

NLRP3炎症通路

The molecular mechanisms underlying sepsis pathogenesis and its regulation are incompletely understood. Here we demonstrated that human AlkB homologue, ALKBH8, plays a critical role in sepsis. we demo

NIAID Data Ecosystem10

IL-1beta activates IL-17 via calcium

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