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The Fun30 ATP-dependent nucleosome remodeler promotes resection of DNA double-strand break ends

ChEBI2012-10-04 更新2026-07-21 收录

DNA损伤修复

染色质重塑

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-38601 数据链接链接失效反馈

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The Fun30 ATP-dependent nucleosome remodeler promotes resection of DNA double-strand break ends

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创建时间：

2012-10-04

DNA损伤修复

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Oxidative DNA damage is important to the evolution of antibiotic resistance: evidence of mutation bias and its medicinal implications

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Several1 studies have revealed that the reactive oxygen species (ROS) induced by antibacterial stimulation accelerates the evolution of antibiotic resistance, which uncovered new links between oxygen

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Chaetomium thermophilum Mre11-Rad50-Nbs1 complex bound to ATPyS (composite structure)

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Chaetomium thermophilum Mre11-Rad50-Nbs1 complex bound to ATPyS (composite structure)

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ARID1A-dependent maintenance of H3.3 is required for repressive CHD4-ZMYND8 chromatin interactions at super-enhancers [12Z_shARID1A_H2A.Z_panH4ac_ChIP]

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Background: SWI/SNF (BAF) chromatin remodeling complexes regulate lineage-specific enhancer activity by promoting accessibility for diverse DNA-binding factors and chromatin regulators. Additionally,

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RNA-seq profiling of human A549 cells with knockdown of chromatin remodeling factor BAZ1A

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Cellular senescence is a well-known cancer prevention mechanism, inducing cancer cells to senescence can enhance cancer immunotherapy. However, how cellular senescence is regulated is not fully unders

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The Fun30 ATP-dependent nucleosome remodeler promotes resection of DNA double-strand break ends

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