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Deregulation of miRNAs in Infant Outflow Tract Myocardium of Non-syndromic Tetralogy of Fallot

ChEBI2012-08-14 更新2026-07-21 收录

先天性心脏病机制

微小RNA

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-40128 数据链接链接失效反馈

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Deregulation of miRNAs in Infant Outflow Tract Myocardium of Non-syndromic Tetralogy of Fallot

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创建时间：

2012-08-14

微小RNA

基因调控

MicroRNAs (miRNAs) are critical to proliferation, differentiation, and development. Here, we characterize gene expression in murine Dicer-null adult mesenchymal stem cell lines, a fibroblast cell type

干细胞与再生医学数据中心2022-02-20 更新130

Table_1_miR-484: A Potential Biomarker in Health and Disease.docx

微小RNA

肿瘤分子机制

Disorders of miR-484 expression are observed in cancer, different diseases or pathological states. There is accumulating evidence that miR-484 plays an essential role in the development as well as the

NIAID Data Ecosystem30

Citrus sinensis Raw sequence reads

微小RNA

甜橙分子生物学

The microRNA of sweet orange

NIAID Data Ecosystem20

Decoding the Long Noncoding RNA during Cardiac Maturation: a Roadmap for Functional Discovery [RNA]. Mus musculus

发育转录组学

先天性心脏病机制

Cardiac maturation during perinatal transition of heart is critical for functional adaptation to hemodynamic load and nutrient environment. Perturbation in this process has major implications in conge

NIAID Data Ecosystem60

The thyroid hormone clearing type 3 deiodinase protects from cardiac congenital abnormalities. The thyroid hormone clearing type 3 deiodinase protects from cardiac congenital abnormalities

器官发育转录组

先天性心脏病机制

We examined the impact of thyroid hormone excess on the mouse fetal heart utilizing a model of deficiency in DIO3, the enzyme that is responsible for clearing thyroid hormones Overall design: RNA sequ

NIAID Data Ecosystem30

Deregulation of miRNAs in Infant Outflow Tract Myocardium of Non-syndromic Tetralogy of Fallot

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