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Cells following mtDNA loss, or NAR1 repression
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创建时间：
2010-06-21
相关数据集
Parkinson Phenotype in Aged PINK1-Deficient Mice Is Accompanied by Progressive Mitochondrial Dysfunction in Absence of Neurodegeneration
BackgroundParkinson's disease (PD) is an adult-onset movement disorder of largely unknown etiology. We have previously shown that loss-of-function mutations of the mitochondrial protein kinase PINK1 (
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Determination of mitochondrial functions and damage in kidney in female LeeSung minipigs with a high-fat diet-induced obesity
We successfully established a DIO-related kidney injury in LeeSung minipigs. In addition, an ATP deficit and oxidative stress were observed in the renal cortex of DIO pigs. Our results provided additi
Mendeley Data2021-06-25 更新20
Data_Sheet_1_Mitochondrial Dysfunction Is an Early Consequence of Partial or Complete Dystrophin Loss in mdx Mice.pdf
Duchenne muscular dystrophy (DMD) is characterized by rapid wasting of skeletal muscle. Mitochondrial dysfunction is a well-known pathological feature of DMD. However, whether mitochondrial dysfunctio
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The Yeast Complex I Equivalent NADH Dehydrogenase Rescues pink1 Mutants
Pink1 is a mitochondrial kinase involved in Parkinson's disease, and loss of Pink1 function affects mitochondrial morphology via a pathway involving Parkin and components of the mitochondrial remodeli
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Ketamine Causes Mitochondrial Dysfunction in Human Induced Pluripotent Stem Cell-Derived Neurons
Purpose Ketamine toxicity has been demonstrated in nonhuman mammalian neurons. To study the toxic effect of ketamine on human neurons, an experimental model of cultured neurons from human induced plur
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