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Mitofusin 2 expression in muscle decreases during aging and generates a gene signature characteristic of aged muscle

ChEBI2016-08-08 更新2026-07-12 收录

肌肉衰老机制

线粒体功能基因标记

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-71501 数据链接链接失效反馈

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资源简介：

Mitofusin 2 expression in muscle decreases during aging and generates a gene signature characteristic of aged muscle

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创建时间：

2016-08-08

肌肉衰老机制

线粒体质量控制

Mitochondrial fusion and fission proteins regulate mitochondrial quality control and mitochondrial metabolism. In turn, mitochondrial dysfunction is associated with aging, although its causes are stil

NIAID Data Ecosystem100

Circular RNAs in monkey muscle: age-dependent changes. Macaca mulatta

环状RNA表达

肌肉衰老机制

Here, we describe the identification of circRNA in monkey (Rhesus macaque) skeletal muscle. RNA sequencing analysis was employed to identify and annotate ~12,000 circRNAs, including numerous circular

NIAID Data Ecosystem80

Ribosome profiling analysis of aging human skeletal muscle (Ribo-Seq). Ribosome profiling analysis of aging human skeletal muscle (Ribo-Seq)

翻译组学

肌肉衰老机制

With advancing age, human muscle loses strength and function, but the molecular causes of these losses are unknown. Skeletal muscle shows an age-dependent decline in the levels of different proteins,

NIAID Data Ecosystem30

Senolytics rescue blunted muscle hypertrophy in old mice

肌肉衰老机制

肌肉发育调控

With aging, skeletal muscle plasticity is attenuated in response to exercise. Here, we report that senescent cells, identified using senescence markers senescence-associated β-Galactosidase (SA β-Gal)

NIAID Data Ecosystem80

A skeletal-muscle senescence blueprint defines an aged-like inflamed niche that inhibits regeneration over lifetime [ATAC-seq]

肌肉衰老机制

ATAC-seq分析

A new sorting protocol was used to identify and isolate different senescent cell types from damaged muscles of young and geriatric mice. Analysis revealed conservation of two universal senescence hall

NIAID Data Ecosystem30

Mitofusin 2 expression in muscle decreases during aging and generates a gene signature characteristic of aged muscle

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