Identification of resistance mechanisms to mTORC1 inhibitor, rapamycin, in an in vivo TNBC model by RNA-seq.

NIAID Data Ecosystem30

Reciprocal inhibition of NOTCH and SOX2 shapes tumor cell plasticity and therapeutic escape in triple negative breast cancer.

三阴性乳腺癌耐药性

癌细胞可塑性

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

NIAID Data Ecosystem20

Conjugates Derived from Lapatinib Derivatives with Cancer Cell Stemness Inhibitors Effectively Reversed Drug Resistance in Triple-Negative Breast Cancer

三阴性乳腺癌耐药性

靶向药物开发

Increasing evidence indicates that the cancer stem cell (CSC) subpopulation contributes to the therapeutic resistance and metastasis of tumors, leading to patient recurrence and death. Herein, we desi

Figshare2021-08-26 更新20

Enhancer reactivation mediates adaptive resistance to FGFR inhibitors in triple-negative breast cancer. Enhancer reactivation mediates adaptive resistance to FGFR inhibitors in triple-negative breast cancer

资源简介：

Enhancer reactivation mediates adaptive resistance to FGFR inhibitors in triple-negative breast cancer. Enhancer reactivation mediates adaptive resistance to FGFR inhibitors in triple-negative breast cancer

资源简介：

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