Loss of RUNX1 favors mesenchymal stem cells development at the expense of hematopoiesis in hPSC-derived hematopoiesis
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https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE232147
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Our data demonstrated an unprecedented role of RUNX1 in regulating hematopoietic and mesenchymal fate decisions in human pluripotent stem cells (hPSC)-derived hematopoiesis We designed an alternative fluorescent gene tdTomato (tdT) and venus reporter and targeted into the non-coding region of exon 3 of RUNX1 gene, which is adjacent to the proximal promoter of RUNX1 gene, using TALEN-mediated homologous recombination
创建时间：
2024-05-17
