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IL-21 regulates the expression of chemokine genes in B-chronic lymphocytic leukemia

ChEBI2013-09-01 更新2026-07-21 收录

白血病分子机制

细胞因子调控

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-42158 数据链接链接失效反馈

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资源简介：

IL-21 regulates the expression of chemokine genes in B-chronic lymphocytic leukemia

应用场景：

创建时间：

2013-09-01

白血病分子机制

基因表达谱分析

Background: MLL (KMT2A)-EB1 (MAPRE1) fusion was identified in a patient with de novo pro-B acute lymphoblastic leukemia. To investigate the leukemogenesis of MLL-EB1 fusion, a retroviral transduction

NIAID Data Ecosystem100

GO:0032736 (positive regulation of interleukin-13 production)

细胞因子调控

生物通路挖掘

This network was assembled automatically by INDRA (http://indra.bio) by processing all available biomedical literature with multiple machine reading systems, and integrating curated pathway databases.

NIAID Data Ecosystem60

DUX4-r neomorphic activity depending on GTF2I in acute lymphoblastic leukemia [RNA-seq]

白血病分子机制

转录组靶点鉴定

Translocations producing rearranged versions of the transcription factor DUX4 (DUX4-r) are one of the most frequent causes of B-cell acute lymphoblastic leukemia (B-ALL). DUX4-r retains the DNA bindin

NIAID Data Ecosystem40

TAF1 is critical for AE driven leukemogenesis [RNA-seq]

白血病分子机制

RNA-seq基因表达

TAF1 is essential for AE driven leukemogenesis. Depletion of TAF1 impairs the recruitment of AE to its target genes, interfering with its control of gene expression. The bromodomains of TAF1 associate

NIAID Data Ecosystem80

EGAS00001000058-sc-20130718a - samples

白血病分子机制

基因组学测序

The ETV6-RUNX1 fusion gene, found in 25% of childhood acute lymphoblastic leukemia (ALL), is acquired in utero but requires additional somatic mutations for overt leukemia. We used exome and low-cover

NIAID Data Ecosystem40

IL-21 regulates the expression of chemokine genes in B-chronic lymphocytic leukemia

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