In this study, we investigated the role of LIN28 in intestinal tumor initiation and invasive progression. We generated animal models with just intestinal LIN28B overexpression, or in combination with
Mouse models of prolactinoma can be useful to better understand molecular mechanisms involved in abnormal lactotroph cell proliferation and secretion. We have previously developed a prolactin receptor
1000, 1500, 12000, and 30000 CD34+CD2+CD7+Lin- and CD34+CD2+CD7-Lin- cells were sorted from T-ALL patient samples (Patients 03, 05, 11) and transplanted into neonatal immune deficient (RAG2−/−γc−/−) m
