官方服务:
资源简介:
22Rv1_AR_ENZ3w_1nM-DHT_rep1
应用场景:
创建时间:
2023-03-09
相关数据集
PC3 cells gene expression after p-coumaric acid treatment. PC3 cells gene expression after p-coumaric acid treatment
Whole genome expression monitoring of human adenocarcinoma prostate cancer (PC3) cell line, after sub-lethal treatment with p-coumaric acid Overall design: Treated vs untreated (control) cells, grown
NIAID Data Ecosystem50
Different doses of androgens induce the expression of distinct gene expression programs (ATAC-Seq). Different doses of androgens induce the expression of distinct gene expression programs (ATAC-Seq)
Most prostate cancers express the androgen receptor (AR), and tumor growth and progression are facilitated by exceptionally low levels of systemic or intratumorally produced androgens. Paradoxically,
NIAID Data Ecosystem40
GREB1 amplifies androgen receptor output in prostate cancer and contributes to antiandrogen resistance. GREB1 amplifies androgen receptor output in prostate cancer and contributes to antiandrogen resistance
Genomic amplification of the androgen receptor (AR) is an established mechanism of antiandrogen resistance in prostate cancer. Here we show that the magnitude of AR signaling output, independent of AR
NIAID Data Ecosystem70
Androgen-starved LNCaP cells treated with gedunin and androgen, androgen alone, or vehicle alone for 24h
Gedunin is a natural product that affects LNCaP androgen-signalling by 24h We used microarrays to detail the androgen-responsive program of gene expression affected by gedunin treatment at 24h Keyword
NIAID Data Ecosystem50



