NK cells could participate in the pathogenesis process of virus infectious diseases through the inhibitory receptor CD94/NKG2A interacting with HLA-E/virus-derived peptide complex. However, the effect
Percentages of IFN-γ producing CD56+/CD3− NK cells and CD56+/CD3+ NK-like T cells for each of eight donors after stimulation of CD56+ cells with the indicated cytokines for 48 h.
