Epigenetically-controlled tumor antigens derived from splice junctions between exons and transposable elements [ChIP-seq]
收藏NIAID Data Ecosystem2026-03-14 收录
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资源简介：
To analyze regulation of JET expression, we generated Setdb1-deficient MC38 and B16OVA cells, using lentivirus-based CRISPR/Cas9. Ctrl and KO cells lines were used to perform ChIP-seq against H3K9me3 and H3K9Ac, with 3 biological replicates. We analyzed H3K9me3 and H3K9Ac signals, in particular on loci involved in forming junctions between exons and TEs.
创建时间：
2023-02-28
