Drug resistance is a central problem in cancer therapy, yet the underlying mechanisms remain obscure. Cancer cells typically acquire resistance via genetic mutations, but may also adopt reversible epi
the cell viability after addition of white wheat glutenexhibited a noteworthy enhancement after a 24-hour period across all concentrations,particularly at a concentration of 200ug/ml where the viabili
Identifying resistance mutations in a drug target provides crucial information. Lentiviral transduction creates multiple types of mutations due to the error-prone nature of the HIV-1 reverse transcrip
