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Splicing function of mitotic regulators links R-loop mediated DNA damage to tumor cell killing

ChEBI2015-04-11 更新2026-07-21 收录

癌症分子机制

DNA损伤响应

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-67770 数据链接链接失效反馈

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Splicing function of mitotic regulators links R-loop mediated DNA damage to tumor cell killing

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创建时间：

2015-04-11

癌症分子机制

依赖性基因调控

Additional file 3. Overexpression of ALKBH5 decreased the proliferation capacity in 2 HCC cell lines, while co-overexpressed of PAQR4 reverted this phenomenon (A and B). Overexpression of ALKBH5 decre

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Role of FGF-2 induced non-coding RNA in cancer

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非编码RNA功能

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

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Aberrant accumulation of Kras-dependent pervasive transcripts during tumor progression renders cancer cells dependent on PAF1 expression. Aberrant accumulation of Kras-dependent pervasive transcripts during tumor progression renders cancer cells dependent on PAF1 expression

癌症分子机制

转录组异常分析

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series

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WTX overexpression effect on gastric cell line

癌症分子机制

肿瘤抑制基因

We used microarrays to detail the global gene expression in AGS gastric cancer cells overexpressing WTX, a tumor suppressor frequently inactivated in Wilms tumor. Results provide insight into roles fo

NIAID Data Ecosystem30

DataSheet4_PRR11 promotes cell proliferation by regulating PTTG1 through interacting with E2F1 transcription factor in pan-cancer.zip

癌症分子机制

细胞周期调控

The upregulated proline rich 11 (PRR11) plays a critical role in cancer progression. The relevant biological functions of PRR11 in pan-cancer development are not well understood. In the current study,

NIAID Data Ecosystem30

Splicing function of mitotic regulators links R-loop mediated DNA damage to tumor cell killing

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