Integrative analysis of liver-specific noncoding regulatory variants associated with the risk of coronary artery disease. Integrative analysis of liver-specific noncoding regulatory variants associated with the risk of coronary artery disease
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We performed promoter Capture Hi-C in HepG2 to investigate interactions between gene promoters and distal elements as a transcription-regulating mechanism contributing to these phenotypes. We also performed ChIP-Seq at 2h, 8h and 23h timepoints in HepG2 for Il1B treatment. Overall design: Promoter Capture Hi-C and Chip-Seq library was produced from HepG2
创建时间：
2020-09-01
