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Persistent Alterations in the imprinted miRNA and snoRNA populations in mouse models of fetal alcohol spectrum disorders (FASD)

ChEBI2011-12-14 更新2026-07-21 收录

胎儿酒精谱系障碍

印记非编码RNA表达谱

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-34413 数据链接链接失效反馈

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资源简介：

Persistent Alterations in the imprinted miRNA and snoRNA populations in mouse models of fetal alcohol spectrum disorders (FASD)

应用场景：

创建时间：

2011-12-14

基因-环境交互

胎儿酒精谱系障碍

P- value for interaction between number of risk alleles and drinking during pregnancy = 0.009 *Total number of risk alleles in ADH1A rs2866151 rs975833, ADH1B rs4147536 and ADH7 rs284779.

NIAID Data Ecosystem50

Additional file 4 of Hippocampal transcriptome analysis following maternal separation implicates altered RNA processing in a mouse model of fetal alcohol spectrum disorder

胎儿酒精谱系障碍

动物转录组学

Additional file 4: Supplementary Table 3. Genes implicated by transcripts in Module 19 associated with the experimental treatment group and the number of center zone entries in the open field test; GO

Figshare2020-05-16 更新30

Expression data from ethanol and saline exposed mice [miRNA]

胎儿酒精谱系障碍

海马基因表达

Mouse models of Fetal Alcohol Spectrum Disorder can be used to assess molecular changes underlying the disorder. Neonatal ethanol exposure in mice can be used to model third trimester ethanol exposure

NIAID Data Ecosystem20

Table_1_Prenatal Alcohol Exposure Results in Sex-Specific Alterations in Circular RNA Expression in the Developing Mouse Brain.XLSX

胎儿酒精谱系障碍

环状RNA调控

Fetal alcohol spectrum disorders (FASD) are heterogeneous disorders associated with alcohol exposure to the developing fetus that are characterized by a range of adverse neurodevelopmental deficits. D

NIAID Data Ecosystem50

Prenatal alcohol exposure: profiling developmental DNA methylation patterns in central and peripheral tissues

产前酒精暴露表观遗传学

胎儿酒精谱系障碍

Prenatal alcohol exposure (PAE) alters the development of neurobiological systems, leading to lasting neuroendocrine, neuroimmune, and neurobehavioral deficits. Although the etiology of this reprogram

NIAID Data Ecosystem20

Persistent Alterations in the imprinted miRNA and snoRNA populations in mouse models of fetal alcohol spectrum disorders (FASD)

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