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资源简介:
22Rv1_AR_100nM-DHT_rep1
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创建时间:
2023-03-09
相关数据集
Effects of Oct1 inhibition in AR signaling
Prostate cancer is the most common cancer in men and AR downstream signalings promote prostate cancer cell proliferation. Oct1 is an AR interacting partner and regulates the transcriptional activity o
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total RNA-seq from PC-3 (ENCSR648KDM)
PC-3_rep2 For data usage terms and conditions, please refer to http://www.genome.gov/27528022 and http://www.genome.gov/Pages/Research/ENCODE/ENCODE_Data_Use_Policy_for_External_Users_03-07-14.pdf ht
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ERG-mediated coregulator complex formation maintains androgen receptor signaling in prostate cancer [PolII ChIP]
We report the effects of ERG on prostate tumorigenesis, ERG-mediated oncogene addiction, and downstream AR signaling pathways. We determined that ERG facilitates AR-signaling and mediates transformati
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GREB1 amplifies androgen receptor output in prostate cancer and contributes to antiandrogen resistance. GREB1 amplifies androgen receptor output in prostate cancer and contributes to antiandrogen resistance
Genomic amplification of the androgen receptor (AR) is an established mechanism of antiandrogen resistance in prostate cancer. Here we show that the magnitude of AR signaling output, independent of AR
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Additional file 4: of Stress alters the expression of cancer-related genes in the prostate
Statistical evaluation of metastasis gene expression by ANOVA with Bonferroni post-hoc analysis to determine overall effects of our treatment paradigm. For genes in which a significant main effect (F-
Figshare2017-09-06 更新40



