Expression profiling of murine cytogenetic normal p53-/- shPU.1 AML cells following gradual PU.1 restoration. Mus musculus
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下载链接：
https://www.ncbi.nlm.nih.gov/bioproject/PRJNA308837
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资源简介：
We have utilized advanced RNAi technology to dynamically restore endogenous PU.1 expression in p53-deficient AML cells . This causes rapid cell cycle shutdown and myeloid differentiation. PU.1 restoration results in regression and clearance and prolongs survival. Overall design: RNA-seq was performed on AML246 cells isolated from triplicate untreated or triplicate Dox-treated mice.
创建时间：
2016-01-14
