Elucidating the mechanisms underlying H3K18la contributes to NSCLC progression
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https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE245990
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Histone lactylation has been served as a novel epigenetic modification that directly stimulates gene transcription from chromatin. In order to study the regulatory mechanisms of H3K18la in non-small cell lung cancer (NSCLC), we performed ChIP-Seq to identify its target genes in NCI-H1299 cell lines. ChIP-Seq with anti-H3K18la in NCI-H1299 cells
创建时间：
2024-11-05
