ATACseq of 3T3L1 cells undergoing differentiation

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脂肪细胞分化

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https://www.ncbi.nlm.nih.gov/bioproject/PRJNA484811 数据链接链接失效反馈

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Confluency-arrested, undifferentiated 3T3-L1 cells were compared with DHA and Dexamethasone treated cells at 4 hrs to assess the effect of early differentiation on chromatin architecture.

创建时间：

2018-08-06

Additional file 2 of Universal NicE-seq for high-resolution accessible chromatin profiling for formaldehyde-fixed and FFPE tissues

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高通量测序

Additional file 2: Supplementary Table 1: HCT116 UniNicE-Seq matrix for library with different amounts of enzyme. Note 25 and 50 U NicE-seq labeling reactions were incubated at 37 °C at 800 RPM. Suppl

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Tousled-like kinase activity is required for transcriptional silencing and suppression of innate immune signaling [HeLa ATAC-Seq]

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先天免疫信号抑制

The Tousled like kinases 1 and 2 (TLK1/2) control histone deposition through the ASF1 histone chaperones and are regulated by the DNA damage response. Depletion of TLK activity caused replication stre

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Downregulated miR-29a/b/c during Contact Inhibition Stage Promote 3T3-L1 Adipogenesis by Targeting DNMT3A

脂肪细胞分化

表观遗传调控

Differentiation of 3T3-L1 cells into adipocytes involves a highly-orchestrated series of events including contact inhibition (CI), clonal expansion, growth arrest, and terminal differentiation. Recent

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Epigenetic Regulation of Nuclear Lamina-Associated Heterochromatin by HAT1 and the Acetylation of Newly Synthesized Histones [pMEF_ATAC_Seq]

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We report that HAT1 regulates chromatin accessibility of large chromosomal domains. Measuring chromatin accessibility in HAT1 WT and KO pMEFs by ATAC-Seq

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Role of SWI/SNF-Related, Matrix Associated, Actin Dependent Regulator of Chromatin A4 (SMARCA4) core complex in epithelial innate response (ATAC-seq). Role of SWI/SNF-Related, Matrix Associated, Actin Dependent Regulator of Chromatin A4 (SMARCA4) core complex in epithelial innate response (ATAC-seq)

先天免疫记忆表观遗传

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We examine the epigenetic role of the SWI/SNF Related, Matrix Associated, Actin Dependent Regulator of Chromatin A4 (SMARCA4) in the epithelial response to RSV infection. Wild type and SMARCA4 silence

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ATACseq of 3T3L1 cells undergoing differentiation

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