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This SuperSeries is composed of the SubSeries listed below. Refer to individual Series
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2020-11-24
相关数据集
RNPS1 inhibits excessive TNF/TNFR signaling to support hematopoiesis in mice. RNPS1 inhibits excessive TNF/TNFR signaling to support hematopoiesis in mice
Null mutations of spliceosome components or cofactors are homozygous lethal in eukaryotes, but viable hypomorphic mutations provide an opportunity to understand the physiological impact of individual
NIAID Data Ecosystem60
A novel RNA m6A modulator Zc3h13 plays an anchor role in facilitating nuclear RNA methylation and mouse embryonic stem cell self-renewal
N6-methyladenosine (m6A) is an abundant modification in eukaryotic mRNA, regulating mRNA dynamics by influencing mRNA stability, splicing, export and translation. Recent studies discovered m6A methylt
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A compendium of hematopoietic regulators, chromatin modifiers and basal transcription factors occupy CBF-MYH11/RUNX1 target genes
A compendium of hematopoietic regulators, chromatin modifiers and basal transcription factors occupy CBF-MYH11/RUNX1 target genes
ChEBI2013-09-09 更新00
MED16 negatively regulates erythropoiesis and myelopoiesis through modulation of chromatin accessibility. MED16 negatively regulates erythropoiesis and myelopoiesis through modulation of chromatin accessibility
This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series
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Data_Sheet_7_N6-methyladenosine modulation classes and immune microenvironment regulation in ischemic stroke.XLSX
N6-methyladenosine (m6A) modifications play an important role in the differentiation and regulation of immune cells. However, research on m6A in ischemic stroke (IS) is still in its infancy, and their
NIAID Data Ecosystem10
