Hematopoietic cell differentiation is required for initiation of acute myeloid leukemia

ChEBI2014-09-17 更新2026-07-21 收录

急性髓系白血病

造血细胞分化

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Hematopoietic cell differentiation is required for initiation of acute myeloid leukemia

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2014-09-17

RNA sequencing expression analysis of murine MLL-AF9;NRAS acute myeloid leukemia cells silenced for E2-2.

急性髓系白血病

RNA测序

We investigated the role of the transcriptional regulators Id2 and E2-2 (encoded by Tcf4) in the context of MLL-rearranged acute myeloid leukemia (AML). Using an AML mouse model driven by a Tet-off in

干细胞与再生医学数据中心2022-02-20 更新80

Supplementary Table 2 from Disruption of the MYC Superenhancer Complex by Dual Targeting of FLT3 and LSD1 in Acute Myeloid Leukemia

急性髓系白血病

转录因子调控

Transcription Factor Enrichment Analysis of MOLM13 Bulk RNA-Seq (24 Hours After Drug Treatment)

Figshare2023-07-05 更新40

Engineering an inducible leukemia-associated transcription factor enables large-scale ex vivo production of functional human phagocytes - Nanostring data

白血病相关基因

造血细胞分化

RNA expression analysis via NanoString nCounter Analysis System with four different cell types (three biological replicates each) using the myeloid innate immunity panel. Total RNA was isolated with t

NIAID Data Ecosystem00

C/EBPalpha regulated microRNA expression profiling in inducible K562-C/EBPalpha-ER cells

急性髓系白血病

MicroRNA调控

C/EBPalpha is an important myeloid transcription factor that is inactivated in approximately 50% of acute myeloid leukemia cases. We were interested in microRNAs that are regulated by C/EBPalpha durin

NIAID Data Ecosystem60

Commonly altered genomic regions in acute myeloid leukemia are enriched for somatic mutations involved in chromatin-remodeling and splicing. Homo sapiens

急性髓系白血病

染色质重塑与剪接突变

Acute myeloid leukemia (AML) is characterized by molecular heterogeneity. As commonly altered genomic regions point to candidate genes involved in leukemogenesis, we used microarray-based comparative

NIAID Data Ecosystem20

Hematopoietic cell differentiation is required for initiation of acute myeloid leukemia

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