首页/数据集/Endoplasmic Reticulum-Resident Protein Sec62 mediated MAPK Signaling Activation drives Colorectal Cancer Metastasis

Endoplasmic Reticulum-Resident Protein Sec62 mediated MAPK Signaling Activation drives Colorectal Cancer Metastasis

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结直肠癌转移机制

Sec62蛋白信号通路

数据链接：

https://www.ncbi.nlm.nih.gov/sra/SRP350689 数据链接链接失效反馈

官方服务：

资源简介：

to investigate Sec62 regulated genes in colon cancer

应用场景：

创建时间：

2021-12-15

结直肠癌转移机制

肝脏lncRNA表达谱

We performed a genome-wide analysis of lncRNA expression to identify novel targets for the further study of liver metastasis in CRC. Samples obtained from CRC patients were analyzed using Arraystar hu

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Additional file 3 of HDAC2 inhibits EMT-mediated cancer metastasis by downregulating the long noncoding RNA H19 in colorectal cancer

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表观遗传与非编码RNA

Additional file 3.

Figshare2024-02-14 更新60

Supplementary Table S4 from GPx2 Suppression of H2O2 Stress Links the Formation of Differentiated Tumor Mass to Metastatic Capacity in Colorectal Cancer

结直肠癌转移机制

肿瘤分子特征

Supplementary Table S4. The epithelial signature.

Figshare2014-11-15 更新40

Complex Behavior of ALDH1A1 and IGFBP1 in Liver Metastasis from a Colorectal Cancer

结直肠癌转移机制

肿瘤基因功能验证

Using our data set (GSE50760) previously established by RNA sequencing, the present study aimed to identify upregulated genes associated with colorectal cancer (CRC) liver metastasis (CLM) and verify

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A medical records based study for KLK8 promotes the proliferation and metastasis of colorectal cancer via the activation of EMT associated with PAR1

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KLK8基因功能

Interventions: Case series:No Primary outcome(s): KLK8 protein concentration Study Design: Single arm

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