Additional file 14 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations

Additional file 14: Table S6 (A) The overlapping genes between differentially expressed genes using DESeq2 and the top 2,000 genes that discriminate HCM from control hearts using O2PLS. (B) Enriched GO terms and pathways by the overlapping genes. (C) Enriched GO terms and pathways by a subset of the overlapping genes, which showed the same changing direction at their protein levels in HCM hearts when compared with controls

附加文件14：表S6（A）使用DESeq2筛选出的差异表达基因与通过O2PLS方法区分肥厚型心肌病（HCM）与对照组心脏的前2000个基因的重叠基因。（B）重叠基因所富集的GO术语和通路。（C）重叠基因子集所富集的GO术语和通路，这些基因在HCM心脏中与对照组相比，其蛋白质水平表现出相同的变化趋势。