Ino80 chromatin remodeling complex promotes recovery of stalled replication forks
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SWI/SNF chromatin remodeling complex is required for initiation of sex-dependent differentiation in mouse germline
Sexual reproduction involves the creation of sex-dependent gametes, oocytes and sperm. In mammals, sexually dimorphic differentiation commences in the primordial germ cells (PGCs) in embryonic gonads;
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PBRM1-deficient PBAF complexes target de novo genomic loci to activate pro-survival genes in clear cell renal cell carcinoma (BRG1 dataset)
PBRM1 is lost in 40% of clear cell renal cell carcinomas (ccRCC) and the combined loss of VHL and PBRM1 drives ccRCC tumorigenesis. PBRM1 is an accessory subunit of the PBAF subclass of the SWI/SNF ch
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ATRX is necessary for cellular senescence and represses HRAS to drive cells from quiescence into senescence [ChIP-Seq]. Homo sapiens
Senescence is a state of stable cell cycle exit that has important implications for development, physiology and disease. It is distinct from quiescence in which cells can be induced to re-enter the ce
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Loss of BAF (mSWI/SNF) complexes causes global transcriptional and chromatin state changes in forebrain development. Mus musculus
We report the genome-wide increase in H3K27 trimethylation (H3K27me3) in murine embryonic cortices as a result of conditional double knock-out (dcKO) of BAF155/BAF170 complex. Overall design: 1) Compa
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INO80 requires a polycomb subunit to regulate the establishment of poised chromatin in murine spermatocytes
This SuperSeries is composed of the SubSeries listed below. Refer to individual Series
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