Telomere dysfucntion activates YAP1 to drive tissue inflammation [ChIP-seq]
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https://www.ncbi.nlm.nih.gov/sra/SRP244587
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资源简介：
Germline telomere maintenance defects provoke inflammatory disease via activated ATM/YAP1/IL-18 signaling in epithelial cells, providing novel therapies for inflammatory conditions associated with short telomeres. Overall design: Telomere proficient intestinal epithelium (G0), which served as control, was compared to the telomere dysfunctional intestinal crypt epithelium (G3). Three samples from each group was pooled and ChIP-seq was performed.
创建时间：
2020-02-29
