Transcription profiling of mouse cell culture treated with LPS to identify of inflammatory genes suppressed by heat shock

ChEBI2010-01-19 更新2026-07-21 收录

炎症基因表达

热激应激响应

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Transcription profiling of mouse cell culture treated with LPS to identify of inflammatory genes suppressed by heat shock

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2010-01-19

Distinct molecular signatures of mild extrinsic and intrinsic atopic dermatitis. Homo sapiens

特应性皮炎分子机制

炎症基因表达

Atopic dermatitis (AD) is a common inflammatory skin disease with underlying defects in epidermal function and immune responses. The goal of this study was to investigate differences in gene expressio

NIAID Data Ecosystem50

Additional file 1 of Bovine oviductal organoids: a multi-omics approach to capture the cellular and extracellular molecular response of the oviduct to heat stress

生殖生物学

热激应激响应

Additional file 1: Table S1. Summary of sequence reads for oviductal organoids mapped to the bovine reference genome.

NIAID Data Ecosystem30

Targeted transcriptomic analysis of C57BL/6 and BALB/c mice during progressive chronic Toxoplasma gondii infection reveals changes in host and parasitic gene expression relating to neuropathology and resolution [Neuroinflammation]. Targeted transcriptomic analysis of C57BL/6 and BALB/c mice during progressive chronic Toxoplasma gondii infection reveals changes in host and parasitic gene expression relating to neuropathology and resolution [Neuroinflammation]

炎症基因表达

弓形虫研究

Toxoplasma gondii is a resilient parasite that infects a multitude of warm-blooded hosts and results in a lifelong chronic infection requiring continuous responses by the host. Chronic infection is ch

NIAID Data Ecosystem30

Inflammatory program of CF-TG cells transfected with cftr gene.

囊性纤维化基因治疗

炎症基因表达

The expression levels of seven commonly regulated genes were determined by qRT-PCR. The results are expressed as the fold changes between CF-TG cells and wt TG cells, and between CF-TG cells transfect

Figshare2015-12-02 更新40

A stimulus-contingent positive feedback loop enables IFN-β dose-dependent activation of pro-inflammatory genes. A stimulus-contingent positive feedback loop enables IFN-β dose-dependent activation of pro-inflammatory genes

干扰素信号通路

炎症基因表达

Type I interferons (IFN) induce powerful anti-viral and innate immune responses via the transcription factor, IFN-stimulated gene factor (ISGF3). However, in some pathological contexts type I IFNs are

NIAID Data Ecosystem30

Transcription profiling of mouse cell culture treated with LPS to identify of inflammatory genes suppressed by heat shock

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