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Additional file 11 of ActIIR inhibition improves motor outcome and preserves muscle fibers after experimental autoimmune neuritis

NIAID Data Ecosystem2026-05-10 收录

自身免疫性神经炎

ActIIR抑制治疗

数据链接：

https://figshare.com/articles/dataset/Additional_file_11_of_ActIIR_inhibition_improves_motor_outcome_and_preserves_muscle_fibers_after_experimental_autoimmune_neuritis/31976939 数据链接链接失效反馈

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资源简介：

Supplementary Material 11.

应用场景：

创建时间：

2026-03-27

自身免疫性神经炎

Th17细胞

Th17 cells are known to exert pathogenic and non-pathogenic functions. Although the cytokine transforming growth factor β1 (TGF-β1) is instrumental for Th17 cell differentiation, it is dispensable for

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Peripheral Blood Transcriptome Analysis of ALS Patients

神经病变转录组学

自身免疫性神经炎

Amyotrophic lateral sclerosis (ALS) and frontotemporal dementia (FTD) are neurodegenerative disorders which overlap in their clinical presentation, pathology, and genetic origin. An over-representatio

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The novel RNA binding protein IMP2 drives a stromal-Th17 cell inflammatory circuit in autoimmune neuroinflammation. The novel RNA binding protein IMP2 drives a stromal-Th17 cell inflammatory circuit in autoimmune neuroinflammation

自身免疫性神经炎

RNA结合蛋白功能

Posttranscriptional control of Ccl2 in stromal cells by IMP2 is required to permit IL-17-driven progression of EAE pathogenesis. Using an optimized data analysis workflow, we mapped about 40 million s

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Activin-A limits Th17 pathogenicity and autoimmune neuroinflammation via CD39 and CD73 ectonucleotidases and Hif1-α -dependent pathways

自身免疫性神经炎

Th17细胞

In multiple sclerosis (MS), Th17 cells are critical drivers of autoimmune central nervous system (CNS) inflammation and demyelination. Th17 cells exhibit functional heterogeneity fostering both pathog

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Identification of a Cxcl10+ monocyte subset with a pathogenic signature in the CNS during auto-immune neuroinflammation

自身免疫性神经炎

单核细胞亚群功能

Multiple Sclerosis (MS) is characterized by pathological inflammation resulting from recruitment of lymphoid and myeloid immune cells from the blood circulation into the central nervous system (CNS).

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Additional file 11 of ActIIR inhibition improves motor outcome and preserves muscle fibers after experimental autoimmune neuritis

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