The cross-talk between prostate cancer cells and myeloid cells to prime cancer metastasis

ChEBI2022-11-30 更新2026-07-12 收录

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肿瘤微环境细胞通讯

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The cross-talk between prostate cancer cells and myeloid cells to prime cancer metastasis

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2022-11-30

Gene expression signatures in anoikis-resistance of human prostate PC-3 cells

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失巢凋亡抵抗基因表达

To investigate potential molecular targets that regulate metastasis and metabolism in anoikis-resistance of prostate cancer cells, we have establised anoikis-resistant prostate PC-3 cells with ultra-l

NIAID Data Ecosystem80

Targeting PRC1 Suppresses Double Negative Prostate Cancer Metastasis by Inhibiting Stemness and Reversing Immune Suppression

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肿瘤免疫微环境

The mechanisms that enable immune evasion at metastatic sites are poorly understood. We show that the Polycomb Repressor Complex-1 drives the colonization of the bones and visceral organs in Double-Ne

NIAID Data Ecosystem50

Akt Mediates Metastasis-Associated Gene 1 (MTA1) Regulating the Expression of E-cadherin and Promoting the Invasiveness of Prostate Cancer Cells

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MTA1-AKT-E-cadherin通路

Human metastasis-associated gene 1 (MTA1) is highly associated with the metastasis of prostate cancer; however, the molecular functions of MTA1 that facilitate metastasis remain unclear. In this study

NIAID Data Ecosystem30

Next generation sequencing showing miRNA profiles of exosomes derived from DHT-treated CAFs and matched ETOH-treated CAFs

外泌体miRNA分析

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Background: Androgen deprivation therapy (ADT) is the backbone of therapy for advanced prostate cancer (PCa). Despite the good initial response, castration resistance and metastatic progression will i

NIAID Data Ecosystem80

Single cell sequencing of mouse syngeneic tumor models. Single cell sequencing of mouse syngeneic tumor models

肿瘤微环境细胞通讯

单细胞转录组学

Tumor ecosystems are composed of multiple cell types that communicate by ligand-receptor interactions. Targeting ligand-receptor interactions, for instance with immune check-point inhibitors, can prov

NIAID Data Ecosystem40

The cross-talk between prostate cancer cells and myeloid cells to prime cancer metastasis

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