Figure 1 - CL patients exhibit high abundance of components of gamma-secretase complex and inflammatory response genes expression in active lesions. Unbiased RNASeq was performed on skin from 7

Figshare2020-12-15 更新60

Table_8_Approaching Inflammation Paradoxes—Proinflammatory Cytokine Blockages Induce Inflammatory Regulators.xlsx

炎症免疫调节

细胞因子网络

The mechanisms that underlie various inflammation paradoxes, metabolically healthy obesity, and increased inflammations after inflammatory cytokine blockades and deficiencies remain poorly determined.

NIAID Data Ecosystem40

Deficiency of p110δ Isoform of the Phosphoinositide 3 Kinase Leads to Enhanced Resistance to Leishmania donovani

利什曼病免疫调节

PI3K磷脂酰肌醇3-激酶

Background Visceral leishmaniasis is the most clinically relevant and dangerous form of human leishmaniasis. Most traditional drugs for treatment of leishmaniasis are toxic, possess many adverse react

NIAID Data Ecosystem60

Khandibharad2022 - IL-12 regulation by IL-10 in Leishmaniasis

利什曼病免疫调节

细胞信号建模

In this model, TLR2-TLR6 mediates MyD88 pathway gets activated, which activates IL-12 production and induces iNOS expression when the Macrophage is infected with Leishmania parasite. The early inducti

NIAID Data Ecosystem50

Khandibharad2022 - IL12 regulating IL10 expression in Leishmaniasis

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