Basal-to-inflammatory transition enhances basal cell carcinoma (BCC) therapy resistance via crosstalk with a pro-inflammatory stromal niche [ATAC-Seq]
收藏NIAID Data Ecosystem2026-05-02 收录
下载链接：
https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE248313
下载链接
链接失效反馈官方服务：
资源简介：
The goal of these ATAC-seq experiments was to determine changes in chromatin accesiblity caused by Il1a and Osm ligand treatment of mouse BCC cells at 48h timepoint versus PBS control There were two experimental conditions in this ATACseq experiment: (1) PBS control and (2) ligand-treated mouse BCC cells. All samples were performed in biological replicates.
创建时间：
2024-10-09
