Journal submission: supplementary tables

Table S1. Summary of six proteomic studies. Table S2. Summary of genome-wide association studies for ten psychiatric disorders. Table S3. Genetic instruments used in Mendelian randomization analyses. Table S4. Summary of cis-pQTLs and LD-proxies used as genetic instruments. Table S5. Full summary statistics of Mendelian randomization analyses. Table S6. Significant Mendelian randomization findings and consistency across different proteomic studies. Table S7. Validations of Mendelian randomization assumptions. Table S8. Full summary statistics of sensitivity analyses. Table S9. Detection of target proteins by antibody profiling in the Brain Atlas. Table S10. Existing drug development efforts involving target proteins documented in the DrugBank. Table S11. Protein-altering variants in high LD with tier 1 protein instruments. Table S12. Results of colocalization analyses. Table S13. Summary of cis-pQTLs demonstrating associations with alternative splicing (sQTL effects). Table S14. Summary of trans-effects of ITIH1 cis-pQTLs. Table S15. Summary of cis-pQTLs demonstrating associations with mRNA abundance (eQTL effects). Table S16. Potential pleiotropic effects of cis-pQTLs identified in the PhenoScanner database. Table S17. Phenome-wide association identified for the <em>SYN2 </em>missense variant rs2923857 in the PhenoScanner database.