An intrachain disulfide-linked IgV-MOG protein elicited chronic atypical EAE in rats.
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aLewis rats were immunized with 50 µg GP69-88 (n = 6) or 200 µg rat IgV-MOG (n = 14) in CFA on day 0. On days 21 and 35, rats were boosted with the same respective antigen (50 µg GP69-88 or 200 µg rat IgV-MOG) in IFA. Immunization and boosting with IgV-MOG were performed with an intrachain disulfide-linked IgV-MOG protein. Rats were scored daily for clinical signs of EAE. Figure 3 and Table 3 portray the same experiment. Recovery of rodents was defined as remission to a clinical score of 0 after an episode of paralytic disease.
An intrachain disulfide-linked IgV-MOG protein elicited chronic atypical EAE in rats.
创建时间：
2014-10-10
