Interaction of Sam68 PxxP peptide mutants with SH3 domains.
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W = wildtype, M1 to M7 = mutants, n/a = not applicable.Compilation of phage-ELISA results indicating changes in binding of the SH3 domains to the PxxP-peptide-mutants compared to the wildtype peptide (++), classified as affinity reduced (+) or interaction abolished (0).Core residues of the PxxP motifs are underlined; introduction of alanine point mutations is highlighted by a black background.
创建时间：
2015-12-02
