首页/数据集/EBF2 links KMT2D-mediated H3K4me1 to suppress pancreatic cancer progression via upregulating KLLN.

EBF2 links KMT2D-mediated H3K4me1 to suppress pancreatic cancer progression via upregulating KLLN.

NIAID Data Ecosystem2026-05-01 收录

癌症表观遗传调控

肿瘤分子机制

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE237366 数据链接链接失效反馈

官方服务：

资源简介：

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

应用场景：

创建时间：

2024-01-29

肿瘤分子机制

miR-520g过表达功能研究

Primitive neuro-ectodermal tumours (PNET) of the supratentorial region are rare, highly malignant embryonal brain tumours affecting young children. We recently highlighted the importance of a microRNA

NIAID Data Ecosystem60

CFTR is a tumor suppressor gene in murine and human intestinal cancer [microarray]

癌症基因组数据

肿瘤分子机制

Analysis of the cystic fibrosis gene Cftr in the colon and small intestine of Cftr-deficient murine model. The hypothesis was loss of Cftr altered expression of genes important in intestinal homeostas

NIAID Data Ecosystem20

Additional file 6 of USP8 promotes the tumorigenesis of intrahepatic cholangiocarcinoma via stabilizing OGT

肝内胆管癌研究

肿瘤分子机制

Supplementary Material 6.

Figshare2024-08-18 更新10

mTORC1 controls cell proliferation by coordinating antagonistic extracellular regulated protein kinase and p38 MAPK signaling

细胞信号传导

肿瘤分子机制

mTOR is a sensor of nutrients/growth factors and a prominent pro-growth signaling molecule; it is highly activated in various tumors and represents a chemotherapeutic target. While mTOR-fueled anaboli

NIAID Data Ecosystem40

AKT dependent NOTCH3 activation drives tumor progression in a model of mesenchymal colorectal cancer subtype. AKT dependent NOTCH3 activation drives tumor progression in a model of mesenchymal colorectal cancer subtype

肿瘤分子机制

结直肠癌亚型分析

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series

NIAID Data Ecosystem10