PRMT5 is essential for sustaining normal adlut hematopoiesis
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PRMT5 is essential for sustaining normal adlut hematopoiesis
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创建时间：
2015-07-15
相关数据集
Transcriptome of Prmt5-deficient and control mouse activated B cells. Transcriptome of Prmt5-deficient and control mouse activated B cells
Quiescent splenic B cells purified from Cg1-cre Prmt5F/F cells and Cg1-cre control mice. Resting B cells were plated on feeder cells expressing CD40L and BAFF and supplemented with IL-4 for activation
NIAID Data Ecosystem70
PRMT5 is essential for sustaining normal adlut hematopoiesis
Examining the effects of PRMT5 loss on the gene expression profile of hematopoietic stem and progenitor cells We established the PRMT5 conditional KO mice, and bred the mice with Mx1 cre transgenic mi
NIAID Data Ecosystem50
PRMT5 is essential for sustaining normal adlut hematopoiesis. Mus musculus
Examining the effects of PRMT5 loss on the gene expression profile of hematopoietic stem and progenitor cells Overall design: We established the PRMT5 conditional KO mice, and bred the mice with Mx1 c
NIAID Data Ecosystem40
PRMT5 is essential for sustaining normal adlut hematopoiesis
Examining the effects of PRMT5 loss on the gene expression profile of hematopoietic stem and progenitor cells Overall design: We established the PRMT5 conditional KO mice, and bred the mice with Mx1 c
NIAID Data Ecosystem70
RNA-seq analysis of PRMT5 targeted genes in Tu212 cells. RNA-seq analysis of PRMT5 targeted genes in Tu212 cells
RNA-sequencing was performed to identify the possible underlying signaling pathway of PRMT5 overexpressing in Tu212 cells. Cells were treated with pEZ-PRMT5 or pEZ-Vector. Overall design: Examination
NIAID Data Ecosystem50
