In meiosis, an excess number of DNA double-strand breaks (DSBs), the initiating DNA lesion, is formed compared to the number of crossovers, one of their repair products that creates the physical links
The files submitted contain calibrated Msh5 ChIP-Seq raw sequence data (50 bp) in the S. cerevisiae SK1 and S288c-sp/YJM789 hybrid. This is continuation of Bioproject PRJNA887541 which contains 150 bp
Additional file 20: Table S11. CTL3.9 SSLP genetic mapping in wild type, recq4a recq4b and HEI10 overexpression lines. Crossover numbers, and cM/Mb values, identified within each interval using Col/Le
Alternative lengthening of telomeres (ALT) is a telomere elongation mechanism observed in ~15% of cancer subtypes. Current models indicate that ALT is mediated by homology-directed repair mechanisms.