Renal cortex expression differences between hypertensive and normotensive patients
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Renal cortex expression differences between hypertensive and normotensive patients
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2011-10-14
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Additional file 5 of Longitudinal serum proteomics identifies inflammatory and metabolic pathways in hypertensive nephrosclerosis progression
Additional file 5.
Figshare2025-05-06 更新80
Isoliensinine ameliorates renal injury via the TGF-β1/SMAD2/3 pathway in spontaneously hypertensive rats [kidney tissue]. Isoliensinine ameliorates renal injury via the TGF-β1/SMAD2/3 pathway in spontaneously hypertensive rats [kidney tissue]
The goals of this study is to compare the differently expressed genes in renal tissues of WKY and SHR with or without isoliensinine treatment. The SHRs (n=12) were randomly divided into 2 groups: SHR,
NIAID Data Ecosystem50
Supplementary Material for: Proteomics to metabolomics: A new insight into the pathogenesis of hypertensive nephropathy
Abstract Background: Hypertensive nephropathy (HN) is a high burden disorder and a leading cause of end-stage renal disorder. In spite of huge investigations, the underlying mechanisms are yet largely
NIAID Data Ecosystem30
Expression data from kidney cortex of control and transgenic sgk1 mice under DOCA/NaCl treatment. Expression data from kidney cortex of control and transgenic sgk1 mice under DOCA/NaCl treatment
Serum and glucocorticoid-induced kinase 1 (SGK1) regulates blood pressure and has been implicated in inflammation and fibrosis. We have tested whether increased SGK1 activity potentiates mineralocorti
NIAID Data Ecosystem70
RNA Sequencing reveals induction of specific renal inflammatory pathways in a rat model of malignant hypertension
In malignant hypertension, far more severe kidney injury occurs than in the âbenignâ form of the disease. The role of high blood pressure and the renin-angiotensin-aldosterone system are well reco
NIAID Data Ecosystem40
