The mouse model with Foxg1 frameshift mutation uncovers the pathophysiology of FOXG1 syndrome

To investigate the gene expression patterns in Foxg1 frameshift mutation, we performed RNAseq in P1 cortices of Q84Pfs heterozygote and littermate wild type control mice. Overall design: Comparative gene expression profiling analysis of RNA-seq data for mouse cortices in wt and FoxG1 KI mutant Q84Pfs.