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Metformin and 2-Deoxyglucose Collaboratively Suppress Human CD4+ T Cell Inflammatory Functions and Activation-Induced Metabolic Reprogramming

NIAID Data Ecosystem2026-03-12 收录

细胞代谢重编程

抗炎免疫药理

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE144354 数据链接链接失效反馈

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资源简介：

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

应用场景：

创建时间：

2020-09-14

细胞代谢重编程

p53信号通路调控

Low glucose metabolite 3-phosphoglycerate switches PHGDH from serine synthesis to p53 activation to control cell fate

integrated proteome resources70

Octopamine metabolically reprograms astrocytes to confer neuroprotection against α-synuclein

α-突触核蛋白

细胞代谢重编程

Octopamine is a well-established invertebrate neurotransmitter involved in fight-or-flight responses. In mammals, its function was replaced by norepinephrine. Nevertheless, it is present at trace amou

NIAID Data Ecosystem40

Cbx4 suppresses CD8+ T cell anti-tumor immunity by reprogramming glycolysis metabolism

细胞代谢重编程

肿瘤免疫治疗

Cbx4 is involved in the regulation of CD8+ T cell metabolism in tumor tissues, which provides a new insight into T cell effector functions and tumor immunotherapy.

NIAID Data Ecosystem10

Mitohormesis reprograms macrophage metabolism to enforce tolerance [vWAT_mac_RNAseq]. Mitohormesis reprograms macrophage metabolism to enforce tolerance [vWAT_mac_RNAseq]

细胞代谢重编程

免疫耐受机制

Macrophages generate mitochondrial reactive oxygen and electrophilic species (mtROS, mtRES) as antimicrobials during Toll-like receptor (TLR)-dependent inflammatory responses. Whether mitochondrial st

NIAID Data Ecosystem40

Table_2_Norbergenin prevents LPS-induced inflammatory responses in macrophages through inhibiting NFκB, MAPK and STAT3 activation and blocking metabolic reprogramming.xlsx

抗炎

细胞代谢重编程

Inflammation is thought to be a key cause of many chronic diseases and cancer. However, current therapeutic agents to control inflammation have limited long-term use potential due to various side-effe

NIAID Data Ecosystem40

Metformin and 2-Deoxyglucose Collaboratively Suppress Human CD4+ T Cell Inflammatory Functions and Activation-Induced Metabolic Reprogramming

资源简介：

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