Influenza A/Hong Kong/156/1997(H5N1) virus NS1 gene mutations F103L and M106I both increase IFN antagonism, virulence and cytoplasmic localization but differ in binding to RIG-I and CPSF30

ChEBI2013-06-22 更新2026-07-21 收录

病毒致病机制

流感病毒功能基因组学

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-48200 数据链接链接失效反馈

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资源简介：

Influenza A/Hong Kong/156/1997(H5N1) virus NS1 gene mutations F103L and M106I both increase IFN antagonism, virulence and cytoplasmic localization but differ in binding to RIG-I and CPSF30

创建时间：

2013-06-22

Influenza A/Hong Kong/156/1997(H5N1) virus NS1 gene mutations F103L and M106I both increase IFN antagonism, virulence and cytoplasmic localization but differ in binding to RIG-I and CPSF30

资源简介：

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