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This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series
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创建时间：
2023-04-06
相关数据集
CCN2 deficiency in smooth muscle cells triggers cell reprogramming and aggravates aneurysm development. CCN2 deficiency in smooth muscle cells triggers cell reprogramming and aggravates aneurysm development
Vascular smooth muscle cell (VSMC) phenotypic switching is widely recognized as a key mechanism responsible for the pathogenesis of several aortic diseases such as aortic aneurysm. Cellular communicat
NIAID Data Ecosystem90
Actin cytoskeleton regulates functional anchorage-migration switch during T-cadherin-induced phenotype modulation of vascular smooth muscle cells
Vascular smooth muscle cell (SMC) switching between differentiated and dedifferentiated phenotypes is reversible and accompanied by morphological and functional alterations that require reconfiguratio
NIAID Data Ecosystem50
Data underlying the publication "Notch signaling regulates strain-mediated phenotypic switching of vascular smooth muscle cells"
The data and resulting publication show the role of Notch signaling in regulating strain-mediated phenotypic switching of vascular smooth muscle cells. The dataset consists of custom-built scripts for
NIAID Data Ecosystem30
Table_1_Insulin Resistance Promotes the Formation of Aortic Dissection by Inducing the Phenotypic Switch of Vascular Smooth Muscle Cells.XLSX
BackgroundInsulin resistance (IR) plays a key role in the development of type 2 diabetes mellitus (T2DM) and is one of its most important characteristics. Previous studies have shown that IR and T2DM
NIAID Data Ecosystem60
Table4_Global trends and Frontier topics about vascular smooth muscle cells phenotype switch: A bibliometric analysis from 1999 to 2021.DOCX
Objective: Vascular smooth muscle cell phenotype switch (VSMCPS) plays a significant role in vascular remodeling. This study aimed to conduct a bibliometric analysis and visualize the knowledge map of
NIAID Data Ecosystem50
