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Loss of CSL unlocks a hypoxic response and enhanced tumor growth potential in breast cancer cells

ChEBI2016-02-28 更新2026-07-12 收录

乳腺癌分子机制

肿瘤微环境缺氧

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-77308 数据链接链接失效反馈

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Loss of CSL unlocks a hypoxic response and enhanced tumor growth potential in breast cancer cells

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创建时间：

2016-02-28

乳腺癌分子机制

RNA调控网络

BRCA-related miRNA sponge interaction network. The network includes 37076 miRNA sponge interactions. (XLSX 484Â kb)

NIAID Data Ecosystem70

The primer sequence.

乳腺癌分子机制

癌症药物靶点

BackgroundBreast cancer continues to be a predominant cause of female mortality globally, characterized by limited therapeutic options and substantial adverse effects. Artesunate (ART), a traditional

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KCTD15 Is Overexpressed in her2+ Positive Breast Cancer Patients and Its Silencing Attenuates Proliferation in SKBR3 CELL LINE

乳腺癌分子机制

基因功能验证

Scientific research for publishing the article

Mendeley Data2024-01-31 更新40

Shc proteins in DMBA induced breast cancer

乳腺癌分子机制

基因编辑模型

The principal factors that control mammary tumor formation remain insufficiently defined. Shc adaptor proteins play an important role in signaling by three well-established biological markers for prog

NIAID Data Ecosystem70

Dietary ligands diindolylmethane and resveratrol result in diverse ERα signaling not seen after E2 and a subset of diindolylmethane mediated signaling needs concurrent AHR activation.. Dietary ligands diindolylmethane and resveratrol result in diverse ERα signaling not seen after E2 and a subset of diindolylmethane mediated signaling needs concurrent AHR activation.

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受体信号通路

Inhibitory crosstalk between estrogen receptor alpha (ER alpha ) and aryl hydrocarbon receptor (AHR) regulates 17-estradiol (E2)-dependent breast cancer cell signaling. ER alpha and AHR are transcript

NIAID Data Ecosystem40

Loss of CSL unlocks a hypoxic response and enhanced tumor growth potential in breast cancer cells

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